CrossRef 19 Lorenz MG, Reipschlager K, Wackernagel W: Plasmid tr

CrossRef 19. Lorenz MG, Reipschlager K, Wackernagel W: Plasmid transformation of naturally competent Acinetobacter calcoaceticus in non-sterile soil extract and

groundwater. Arch selleck products Microbiol 1992,157(4):355–360.PubMedCrossRef 20. Berge M, Mortier-Barriere I, Martin B, Claverys JP: Transformation of Streptococcus pneumoniae relies on DprA- and RecA-dependent protection of incoming DNA single strands. E7080 research buy Mol Microbiol 2003,50(2):527–536.PubMedCrossRef 21. Mortier-Barriere I, Velten M, Dupaigne P, Mirouze N, Pietrement O, McGovern S, Fichant G, Martin B, Noirot P, Le Cam E, Polard P, Claverys JP: A key presynaptic role in transformation for a widespread bacterial protein: DprA conveys incoming ssDNA to RecA. Cell 2007,130(5):824–836.PubMedCrossRef 22. Meibom KL, Li XB, Nielsen AT, Wu CY, Roseman S, Schoolnik GK: The Vibrio cholerae chitin utilization program. Proc Natl Acad Sci USA 2004,101(8):2524–2529.PubMedCrossRef 23. Palmen R, Hellingwerf KJ: Uptake and processing of DNA by Acinetobacter calcoaceticus

–a review. Gene 1997,192(1):179–190.PubMedCrossRef 24. Pifer ML, Smith HO: Processing of donor DNA during Haemophilus influenzae transformation: analysis using a model plasmid system. Proc Natl Acad Sci USA 1985,82(11):3731–3735.PubMedCrossRef 25. Goodman SD, Scocca JJ: Factors influencing the specific interaction of Neisseria gonorrhoeae CP673451 cost with transforming DNA. J Bacteriol 1991,173(18):5921–5923.PubMed 26. Smith HO, Gwinn ML, Salzberg SL: DNA uptake signal sequences in naturally transformable bacteria. Res Microbiol 1999,150(9–10):603–616.PubMedCrossRef 27. Stein DC: Transformation Ketotifen of Neisseria gonorrhoeae: physical requirements of the transforming DNA. Can J Microbiol 1991,37(5):345–349.PubMedCrossRef 28. Smith HO, Tomb JF, Dougherty BA, Fleischmann RD, Venter JC: Frequency and distribution

of DNA uptake signal sequences in the Haemophilus influenzae Rd genome. Science 1995,269(5223):538–540.PubMedCrossRef Authors’ contributions RLM contributed intellectually to this study, carried out experiments, and analyzed data. MB served as principal investigator, designed and coordinated the study, performed experiments, analyzed data, and wrote the manuscript. All authors read and approved the manuscript.”
“Background Lyme disease is a multisystemic zoonotic disease caused by Borrelia burgdorferi sensu lato (B. burgdorferi s. l.). B. burgdorferi s. l. circulates in an enzootic cycle between the primary vertebrate reservoir and the ticks[1, 2]. A wide range of mammals are severeded as reservoir hosts in the natural cycle of B. burgdorferi sensu lato[3, 4]. Different species of rodents, mainly mice and voles, are identified to be efficient natural reservoirs for B. burgdorferi sensu lato. They could naturally infect B. burgdorferi sensu lato and remain infective for a long time.

Comments are closed.